1) 3T3-L1 cells
3T3-L1细胞
1.
The role of galanin in resistin gene expression in rat adipose tissue and 3T3-L1 cells;
在大鼠脂肪组织及3T3-L1细胞中甘丙肽对抵抗素基因表达的影响
2.
Methods The cultured 3T3-L1 cells were pretreated with naringin,and then MTT method was used to detect the proliferation of the cells,oil red O staining method and spectrophotography were applied to analyze the degree of differentiation.
方法培养3T3-L1细胞,并用不同浓度的柚皮苷进行干预,以四甲基偶氮唑盐(MTT)法检测细胞增殖,用油红O染色和染色比色法分析脂肪细胞的分化程度。
2) 3T3-L1 cell
3T3-L1细胞
1.
Insulin up-regulates expression of apelin gene in 3T3-L1 cells:a preliminary observation;
胰岛素上调3T3-L1细胞apelin基因表达的初步观察
2.
Effects of recombinant adenovirus-mediated siRNA on adiponectin expression in 3T3-L1 cells;
腺病毒载体介导的siRNA对3T3-L1细胞脂联素表达的影响
3.
Ten novel compounds were designed and synthesized on the basis of compound 1,their insulin-sensitizing activities were evaluated in 3T3-L1 cells.
结果表明化合物10在3T3-L1脂肪细胞模型上表现出较强的促3T3-L1细胞分化活性,提示其可能具有较好的胰岛素增敏作用。
3) 3T3-L1
3T3-L1细胞
1.
Meanwhile,3T3-L1 cells were used and incubated respectively with arachid.
同时进行了PUFAs对体外培养3T3-L1细胞分化的干预实验,在细胞被诱导分化D5,在培养基中分别加入油酸(OA,C18:1n-9)、花生四烯酸(AA,C20:4n-6)、二十碳五烯酸(EPA,C20:5n-3)和二十二碳六烯酸(DHA,22:6n-3,0。
4) 3T3-L1 adipocytes
3T3-L1脂肪细胞
1.
Molecular mechanism of insulin resistance induced by free fatty acids in 3T3-L1 adipocytes through targeting nuclear-kB p65;
游离脂肪酸刺激核因子NF-kBp65核转位诱导3T3-L1脂肪细胞胰岛素抵抗的分子机制
2.
Expression of adiponutrin mRNA in mature 3T3-L1 adipocytes;
Adiponutrin mRNA在分化成熟的3T3-L1脂肪细胞中的表达
3.
Continuously activated Akt inhibits adiponectin secretion in 3T3-L1 adipocytes;
持续激活的Akt减少了3T3-L1脂肪细胞脂联素蛋白的表达
5) 3T3-L1 preadipocyte
3T3-L1前脂肪细胞
1.
Obestatin inhibits proliferation and differentiation of 3T3-L1 preadipocytes;
Obestatin抑制3T3-L1前脂肪细胞的增殖与分化
2.
Angiopoietin-like protein 4(ANGPTL4) gene expression during the period of 3T3-L1 preadipocyte differentiation
ANGPTL4基因在3T3-L1前脂肪细胞诱导分化过程中表达水平的变化
3.
The differentiation of 3T3-L1 preadipocyte,HL-60,K562 and B16 cells were assessed by microscopy and Oil red O staining,NBT reduction and phagocytosis,haemoglobin(Hb) content and melanin content,respectively.
方法:SRB法和乳酸脱氢酶释放法检测B16细胞活性;油红染色法检测3T3-L1前脂肪细胞的分化;NBT还原能力和细胞吞噬功能法检测HL-60细胞的分化;血红蛋白含量法检测K562细胞的分化;黑色素含量法检测B16细胞的分化。
6) 3T3-L1 adipocyte
3T3-L1脂肪细胞
1.
Establishment of GOD-POD assay in a minimal way and application to glucose metabolism of 3T3-L1 adipocyte and HepG2 cell in vitro;
GOD-POD法微量化测定方法的建立及其在3T3-L1脂肪细胞和HepG2细胞糖摄取中的应用
2.
Akt regulating adiponectin secretion in 3T3-L1 adipocytes;
蛋白激酶B对3T3-L1脂肪细胞脂联素蛋白质表达的调控
3.
This paper establishes the insulin resistance model of 3T3-L1 adipocytes.
建立3T3-L1脂肪细胞IR模型,葡萄糖氧化酶法测定葛根素处理48 h后培养液中葡萄糖残存量、铜试剂显色法测定游离脂肪酸浓度。
补充资料：“痘痕”红细胞
“痘痕”红细胞
“pocked”red blood cell
Koyama及其同道(1962年)首先利用扫描电镜发现切除脾脏患者，有相当比例的红细胞表面显示“空泡”，而正常人有此空泡者少见。Holroyd(1969年)等利用干涉显微镜证实无脾者的许多红细胞的光滑表面出现“凹陷”，形态似“痘痕”，故称为“痘痕”红细胞。此种“痘痕”即扫描电镜下所见的“空泡”。用干涉显微镜计数循环血“痘痕”红细胞百分数，可评价脾功能。将脾功能分3种状态：正常脾功能的痘痕红细胞值为0～0.3%，无脾功能测值为19%～52%，脾功能低下的新生儿测值为0.6%～2.8%。“痘痕”的检测对评价脾功能及进一步开展脾移植和保留脾脏手术的临床实践和科研，是一项有用的、简便有效方法。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条