1) cardiomyocyte hypertrophy
心肌细胞肥大
1.
Effects of exogenous Apelin on cardiomyocyte hypertrophy;
Apelin对心肌细胞肥大的影响
2.
Study of cross-talk between stat family and smads pathway mediated cardiomyocyte hypertrophy in cultured neonatal rat in vitro
新生大鼠心肌细胞通过STAT和Smads信号交叉对话介导心肌细胞肥大的体外研究
3.
OBJECTIVE To observe effects of Sodium tanshinone IIA sulfonate(STS) on angiotensin II(Ang Ⅱ)-induced cardiomyocyte hypertrophy and expressions of phosphorylated p38(p-p38) and mitogen-activated protein kinase phosphatase-1(MKP-1).
目的观察丹参酮ⅡA磺酸钠(Sodium tanshinoneⅡA sulfonate,STS)对血管紧张素Ⅱ(angiotensinⅡ,AngⅡ)诱导的心肌细胞肥大反应及p-p38,MKP-1表达的影响。
2) Cardiac hypertrophy
心肌细胞肥大
1.
The Effect of Erythropoietin on cardiac hypertrophy
促红细胞生成素促心肌细胞肥大的实验研究
2.
Objective: to study the inhibition effect of simvastatin on cardiac hypertrophy induced by myocardial infarction,and to investigate whether simvastatin prevent cardiac hypertrophy through attenuation of RhoA/ROCK and cyclin D1.
目的:观察辛伐他汀对大鼠心肌梗死后心肌细胞肥大的抑制作用,探讨小G蛋白RhoA激酶(ROCK)/细胞周期蛋白D1(Cyclin D1)途径在辛伐他汀抑制心肌细胞肥大中的信号转导作用。
3.
However,little is known about the role of Epo in the development of cardiac hypertrophy.
有关Epo对心肌细胞肥大影响的报导在国内外非常有限。
3) human adult hypertrophic cardiomyocytes
人肥大心肌细胞
1.
The establishment of the model for hypoxia-reoxygenation induced apoptosis of culturing human adult hypertrophic cardiomyocytes;
缺氧/复氧诱导培养人肥大心肌细胞凋亡模型的建立
4) hypertrophic cardiomyocytes
肥大心肌细胞
1.
Objective To investigate the hypertrophic cardiomyocytes apoptosis induced by hypoxia and hypoxia /reoxygenation.
结果肥大心肌细胞在缺氧8h时即出现显著的细胞凋亡。
2.
In the study, we investigated the change rule of the cytoskeleton and biomechanics of hypertrophic cardiomyocytes and furthermore illuminated.
本研究目的就在于观察肥大心肌细胞骨架、生物力学特性的变化规律,并进一步阐明细胞骨架改变对肥大心肌细胞生物力学特性的具体影响及其调节机制。
补充资料：心肌细胞
心肌细胞
myocardium cell
心肌细胞根据其生理功能可分为两类，即工作性心肌细胞和传导性心肌细胞。其中工作性心肌细胞构成心房肌和心室肌，是心壁的主要组成部分，具有兴奋、传导和收缩的功能；而自律性心肌细胞组成心脏的传导系统，包括窦房结、结间束、房室交界、左右束支和普肯耶纤维网。它们除有兴奋、传导的功能外，还有自律性，即不受神经支配与体液的影响，有产生周期性兴奋的能力。窦房结兴奋，将冲动扩布到心肌，引起心肌的收缩，从而产生心肌细胞的生物电现象。所以心肌细胞具有兴奋性、自律性、传导性和收缩性四种生理特性。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条