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1)  IL 2,5 HT 3 receptor
IL-2,5-HT3受体
2)  5-HT3 receptor
5-HT3受体
1.
Objective To study antiemetic mechanism and the clinical application of 5-HT3 receptor antagonists by researching the mechanisms of the chemotherapy-induced vomiting and the characteristics of the 5-HT3 receptors.
目的通过对5-HT3受体特点的研究和5-HT3受体参与的化疗致吐机制的剖析,阐明5-HT3受体拮抗剂对抗化疗致吐方面的作用机制和临床应用。
3)  5-HT3 receptor antagonist
5-HT3受体拮抗剂
1.
Ondansetron Hydrochloride is a high effective and high selective 5-HT3 receptor antagonist and is used on anti-emesis after chemotherapy and radiotherapy and postoperation.
盐酸昂丹司琼(Ondansetron Hydrochloride)是一种高效,高选择性的5-HT3受体拮抗剂[1,2],用于对抗癌症化学治疗和放射治疗引起的呕吐以及手术后的急性呕吐[3-5]。
4)  5-HT3 receptor antagonists
5-HT3受体拮抗药
1.
Objective:To evaluate the current situation and the developing trend of 5-HT3 receptor antagonists used in 25 hospital of Wuhan during the period of 2003~2005 in order to provide reference for clinical doctors and pharmacists.
目的:分析武汉部分医院2003~2005年5-HT3受体拮抗药使用情况及用药趋势,为临床用药提供参考。
5)  5-HT3 receptor blocking agents
5-HT3受体阻断剂
1.
Objective: We explained the investigation of 5-HT3 receptor blocking agents,NK-1 receptor blocking agents,corti-costeroid et al to comprehend nausea and vomiting drugs,which may be helpful for searching and developing safer,more effective and economical drugs to prevent and treat nausea and vomiting.
目的:通过对5-HT3受体阻断剂,NK-1受体阻断剂,皮质类固醇等药物的研究进展进行阐述,了解用于预防恶心呕吐的药物,为预防和治疗化疗后的恶心呕吐反应寻找或开发出更安全、有效、经济的药物,从而减少或避免在化疗过程中的依从性差,疗效不稳定等情况。
6)  IL-2 receptor
IL-2受体
1.
Pilot study on the anti-reject effect of antisense oligodeoxyribonucleic acid of IL-2 receptor;
IL-2受体反义核酸抗排斥反应作用的初步研究
2.
The influence of human red blood cells on the synthesis of DNA and the expression of IL-2 receptor as well as the killing activity of LAK celi were observed by means of 3H-TdR incorporation, APAAP immunostaining and MTT test-ing.
本文用~3H-TdR掺入法、APAAP免疫染色法和MTT法研究人红细胞(RBC)对LAK细胞DNA合成、IL-2受体(IL-2R)表达及杀伤活性的影响。
补充资料:IL-3
分子式:
CAS号:

性质:又称白细胞介素-3。主要由活化T细胞或T细胞克隆产生。含133个氨基酸残基。免疫调节剂,可刺激多能干细胞和多种祖细胞的增殖和分化。刺激皮肤上皮细胞、CD4-CD8-TCRαβ细胞、巨噬细胞、嗜碱性粒细胞的增殖,阻止巨噬细胞发生程序性细胞死亡。

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