1) JAK/STAT path-way
JAK/STAT途径
2) JAK-STAT signal transduction pathway
JAK-STAT信号途径
1.
Here we reported the research advances in type I and II interferon genes and their receptors,the JAK-STAT signal transduction pathway,the interferon regulatory factor family and some interferon-inducible proteins in fish.
本文介绍了鱼类I型和II型干扰素及其受体、干扰素调节因子、JAK-STAT信号途径、干扰素诱导蛋白等的研究进展。
3) JAK-STAT pathway
JAK-STAT信号转导途径
1.
ObjectiveTo investigate the role of interferon inducible protein IFI 16 and JAK-STAT pathway in the pathogenesis of systemic lupus erythematosus(SLE).
体外培养PBMC,用JAK2抑制剂AG490抑制JAK-STAT信号转导途径,再用RT-PCR检测PBMC中IFI 16的表达水平。
2.
Objective To investigate the expression of IFI16 and the function of JAK-STAT pathway in peripheral blood mononuclear cell of patients with systemic lupus erythematosus(SLE).
体外培养PBMC,分别用JAK抑制剂AG490和STAT3的decoy寡核苷酸(ODNs)抑制JAK-STAT信号转导途径,再用RT-PCR检测PBMC中IFI16的表达水平。
4) JAK/STAT pathway
JAK/STAT通路
1.
Several cytokines induce the expression of the suppressor of cytokine signaling(SOCS)gene through JAK/STAT pathway,while SOCS protein negatively regulates signal transduction of cytokine pathway,and a negative feedback loop of cytokine signal transduction is thus completed.
一系列细胞因子通过JAK/STAT通路诱导细胞因子信号转导抑制因子(SOCS)基因的表达,SOCS蛋白又负反馈调节细胞因子信号转导通路,形成细胞因子信号转导反馈调节环。
5) JAK-STAT pathway
JAK-STAT通路
1.
JAK-STAT pathway modulates NF-кB-mediated cytoprotection of H_2O_2 preconditioning;
JAK-STAT通路调制NF-κB介导H_2O_2预处理的细胞保护作用
2.
Two important cell signal pathways,JAK-STAT pathway and.
两条主要的细胞信号转导通路,JAK-STAT通路和MAPK通路,介导调节调节因子对造血相关基因的活性调控。
6) JAK/STAT
JAK/STAT通路
1.
Objective To determine the effect of janus kinase/signal transducer and activator of transcription(JAK/STAT) pathway on the expressions of interferon-γ(IFN-γ) in vital organs of rats with sepsis.
结论腹腔脓毒症时肝、肺IFN-γ表达明显升高,抑制JAK/STAT通路的活化可下调肝、肺IFN-γ的表达,这可能是防止及治疗多器官功能衰竭的一个新的干预途径。
2.
Objective: The purpose of this study was to investigate the effect of janus kinase/signal transducer and activator of transcription (JAK/STAT) pathway on the expression of Interleukin-10(IL-10) in hepatic tissue of rats with sepsis.
结论 :腹腔脓毒症使IL - 10表达明显升高 ,抑制JAK/STAT通路的活化可进一步上调肝组织IL - 10的表。
补充资料：E-D途径
分子式：
CAS号：
性质：又称2-酮-3-脱氧-6-磷酸葡糖酸(KDPG)裂解途径。最早由Entner和Doudoroff在嗜糖假单胞菌(Pseudomonassaccharophila)中发现，故名。是若干缺乏完整EMP途径的微生物所具有的EMP替代途径。其特点是：葡萄糖只经四步反应即可产丙酮酸，关键反应是KDPG裂解成丙酮酸和3-磷酸甘油醛。细菌的酒精发酵就是通过本途径进行的。
CAS号：
性质：又称2-酮-3-脱氧-6-磷酸葡糖酸(KDPG)裂解途径。最早由Entner和Doudoroff在嗜糖假单胞菌(Pseudomonassaccharophila)中发现，故名。是若干缺乏完整EMP途径的微生物所具有的EMP替代途径。其特点是：葡萄糖只经四步反应即可产丙酮酸，关键反应是KDPG裂解成丙酮酸和3-磷酸甘油醛。细菌的酒精发酵就是通过本途径进行的。
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参考词条