1) Fructozyme
果糖酶
2) fructokinase
果糖激酶
1.
The glucose,fructose and sucrose con- tents and the activities of invertase,sucrose syn- thase(SS),sucrose phosphate synthase(SPS),hex- okinase(HXK)and fructokinase(FRK)of‘Tochiotome’strawberry aggregate fruit were ana- lyzed at various stages of fruit development.
己糖激酶和果糖激酶则表现出与糖梯度相反的变化。
2.
Sugars accumulation and fructokinase ac- tivity during satsuma mandarin fruit development in relation to the effect of extra nitrogenous fertilizer on the activity and expression of fructokinase were studied.
研究了温州蜜柑果实发育进程中糖含量变化与果糖激酶活性变化的关系及增施氮肥对果实果糖激酶活性和基因表达的影响。
3.
Two genomic DNA fragments encoding putative fructokinase gene were cloned from Citrus unshiu Marc, using PCR, named Cufrk1 and Cufrk2.
植物果糖激酶(FRK)在果糖磷酸化中起重要作用。
3) Levan hydrolase
果聚糖酶
4) pectosase,pectosinase
果胶糖酶
5) fructosidase
[,fræktə'saideis]
果糖甙酶
补充资料:果糖-1-磷酸醛缩酶缺陷
果糖-1-磷酸醛缩酶缺陷
又称遗传性果糖不耐受症。本症为常染色体隐性遗传,部分杂合子也可有症状。临床主要表现有呕吐、腹泻、黄疸、肝大、生长障碍以及出血、低血糖。果糖代谢主要在肝细胞内,果糖-1-磷酸醛缩酶的缺乏,肝细胞内1-磷酸果糖堆积,后者抑制了果糖-1,6-二磷酸醛缩酶的作用,使正常从甘油、氨基酸等转变成葡萄糖的异生作用受阻,从而产生低血糖。过多的1-磷酸果糖还抑制了磷酸化酶的作用,阻碍了糖原变成葡萄糖,这是引起低血糖的另一个原因。
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