1) peroxisome proliferator-activated receptor-γ
过氧化物酶体增殖子活化受体-γ
1.
Aim:To observe effects of RNAi for the target on peroxisome proliferator-activated receptor-γ(PPARγ) suppress the adipogenic differentiation of bone marrow stromal cells (MSCs) induced by ethanol.
目的:观察靶向过氧化物酶体增殖子活化受体-γ(PPARγ)基因RNA干扰阻断乙醇诱导骨髓基质细胞的成脂分化效应。
2.
Aim:To observe RNA interference(RNAi) for the target on peroxisome proliferator-activated receptor-γ(PPARγ) and suppress the effects of steroid to maintain the osteogenic differentiation of bone marrow stromal cells (MSCs).
目的:观察靶向过氧化物酶体增殖子活化受体-γ(PPARγ)基因RNA干扰阻断激素作用保持骨髓基质细胞(MSCs)成骨分化的作用。
3.
Aim:To observe the effects of inhibition for the target RNAi to peroxisome proliferator-activated receptor-γ(PPARγ)on steroid-induced adipogenesis in marrow stromal cells(MSCs).
目的:观察靶向过氧化物酶体增殖子活化受体-γ(PPARγ)RNA干扰抑制激素诱导骨髓基质细胞成脂分化的作用。
2) peroxisome proliferator-activated receptor γ
过氧化物酶体增殖因子活化受体γ
1.
Aim To study the effect of Curcumin on the apoptosis of hepatic stellate cells (HSC), and the correlation between the effect and peroxisome proliferator-activated receptor γ(PPARγ) signal.
结论姜黄素促进过氧化物酶体增殖因子活化受体γ表达和核转位/重分布,诱导肝星状细胞凋亡。
2.
Objective To study the effect of curcumin on the activity of matrix metalloproteinases(MMPs) and nuclear expression of RelA in rat hepatic stellate cells(HSCs) by activating peroxisome proliferator-activated receptor γ(PPARγ) signal in vitro.
目的研究姜黄素激活过氧化物酶体增殖因子活化受体γ(PPARγ)信号对大鼠肝星状细胞(HSC)活化、基质金属蛋白酶(MMPs)活性和胞核核因子-κBp65(RelA)表达的影响。
3) Peroxisome proliferateerator-activated receptorγ(PPARγ)
过氧化物酶体增殖因子活化受体γ(PPARγ)
4) PAR-2
过氧化物酶体增殖因子活化受体-γ
5) Peroxisome Proli ferator-activated Receptor-γ(PPAR-γ)
过氧化物酶体增殖活化体受体-γ(PPAR-γ)
6) peroxisome proliferator-activated receptors-γ(PPAR-γ)
过氧化物酶体增殖物激活受体γ(PPAR-γ)
补充资料：过氧
过氧
guòyǎng
[peroxy-] 含有过氧物的特征基-o-o-
例:过氧焦硫酸盐
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条